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Examples
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"So now, we have some clear genetic markers that we can go and test to see how they might affect a host of metabolic processes," Caporaso said.
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Now, it's been known for a few decades that this particular CYP1A2 gene is what metabolized caffeine, Caporaso said.
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Caporaso joins colleagues from the Harvard School of Public Health, Brigham and Women's Hospital, the University of North Carolina at Chapel Hill, and others to report these findings in the April issue of PLoS Genetics.
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Pooling all the data, Caporaso and his associates found that those who carried the highest-consumption genotype of either the CYP1A2 or the AHR gene consumed an extra 40 milligrams of caffeine compared with those bearing the lowest-consumption genotype.
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"So now, we have some clear genetic markers that we can go and test to see how they might affect a host of metabolic processes," Caporaso said.
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Now, it's been known for a few decades that this particular CYP1A2 gene is what metabolized caffeine, Caporaso said.
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The point here is that the way we drink caffeine is not just random, said Caporaso, who, like his co-authors, declared no competing interests.
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Pooling all the data, Caporaso and his associates found that those who carried the highest-consumption genotype of either the CYP1A2 or the AHR gene consumed an extra 40 milligrams of caffeine compared with those bearing the lowest-consumption genotype.
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"It's really an incredible story," said study co-author Dr. Neil Caporaso, branch chief of genetic epidemiology at the National Cancer Institute.
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"It's really an incredible story," said study co-author Dr. Neil Caporaso, branch chief of genetic epidemiology at the National Cancer Institute.
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