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Examples
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Oliver and her colleagues set out to study cisplatin resistance in mice with a mutation in a gene called Kras, which leads the animals to develop lung cancer.
PhysOrg.com - latest science and technology news stories PhysOrg Team 2010
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Oliver and her colleagues set out to study cisplatin resistance in mice with a mutation in a gene called Kras, which leads the animals to develop lung cancer.
PhysOrg.com - latest science and technology news stories 2010
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Oliver and her colleagues set out to study cisplatin resistance in mice with a mutation in a gene called Kras, which leads the animals to develop lung cancer.
PhysOrg.com - latest science and technology news stories 2010
-
Oliver and her colleagues set out to study cisplatin resistance in mice with a mutation in a gene called Kras, which leads the animals to develop lung cancer.
PhysOrg.com - latest science and technology news stories PhysOrg Team 2010
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Oliver and her colleagues set out to study cisplatin resistance in mice with a mutation in a gene called Kras, which leads the animals to develop lung cancer.
PhysOrg.com - latest science and technology news stories 2010
-
Oliver and her colleagues set out to study cisplatin resistance in mice with a mutation in a gene called Kras, which leads the animals to develop lung cancer.
PhysOrg.com - latest science and technology news stories 2010
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In experiments in living mice, the team discovered that if p53 is disabled in immature blood-cell "precursors" in which a mutation in a gene called Kras is also present, a built-in braking mechanism fails to engage and the cells proliferate out of control.
innovations-report 2010
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UCSF scientists have discovered how a mutated gene known as Kras is able to hijack mouse cells damaged by pancreatic cancer cells.
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In experiments in living mice, the team discovered that if p53 is disabled in immature blood-cell "precursors" in which a mutation in a gene called Kras is also present, a built-in braking mechanism fails to engage and the cells proliferate out of control.
Science Blog 2010
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In experiments in living mice, the team discovered that if p53 is disabled in immature blood-cell "precursors" in which a mutation in a gene called Kras is also present, a built-in braking mechanism fails to engage and the cells proliferate out of control.
THE MEDICAL NEWS 2010
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