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Examples
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Despite its importance, relatively little is known about the structural determinants of beta-globin mRNA stability or how they contribute to regulating beta-globin gene expression.
Sickle cell disease research at The Children's Hospital 2010
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This project is designed to establish a comprehensive understanding of the structures, factors and mechanisms that affect the stability of human beta-globin mRNA. This information promises to be helpful in designing therapies for sickle cell disease.
Sickle cell disease research at The Children's Hospital 2010
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Kucherlapati, R.S. Insertion of DNA sequences into the human chromosomal beta-globin locus by homologous recombination.
The 2007 Nobel Prize in Physiology or Medicine - Advanced Information 2007
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For example, when Michael and I engaged in debate at the 1995 meeting of the American Scientific Affiliation, I argued that the 100% match of DNA sequences in the pseudogene region of beta-globin was proof that humans and gorillas shared a recent common ancestor.
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We also share the same exact pseudogene in the beta-globin gene cluster as other primates, and as Miller points out, agreeing on errors is a classic example of decisive evidence for plagiarism pp.99-101.
Only A Theory James F. McGrath 2008
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We also share the same exact pseudogene in the beta-globin gene cluster as other primates, and as Miller points out, agreeing on errors is a classic example of decisive evidence for plagiarism pp.99-101.
Archive 2008-08-01 James F. McGrath 2008
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Kan and Andres Dozy published a paper in The Lancet in which they used a variation in the flanking DNA of the beta-globin gene in the first successful prenatal genetic diagnosis of sickle cell anemia.
Junk DNA, Linguistics and the scientific vacuity of Intelligent Design - The Panda's Thumb 2007
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For example, when Michael and I engaged in debate at the 1995 meeting of the American Scientific Affiliation, I argued that the 100% match of DNA sequences in the pseudogene region of beta-globin was proof that humans and gorillas shared a recent common ancestor.
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In different patients, small defects in the genetic material have been found, resulting in errors in the splicing process and thus in the synthesis of poorly functioning beta-globin.
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A normal beta-globin gene is presented in A, and two mutated genes that result in beta-thalassemia are shown in B and C. Arrows mark the position of point mutations.
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