from The American Heritage® Dictionary of the English Language, 4th Edition
- n. Formation of atheromatous deposits, especially on the innermost layer of arterial walls.
from Wiktionary, Creative Commons Attribution/Share-Alike License
- n. The formation of atheromas, especially on the walls of the arteries
from WordNet 3.0 Copyright 2006 by Princeton University. All rights reserved.
- n. the formation of atheromas on the walls of the arteries as in atherosclerosis
Another is its ability to increase blood cholesterol (by modifying enzyme activities) and to enhance atherogenesis, which is the early stage of cardiovascular disease.
So these latest results suggest that the current thinking on atherogenesis is completely misguided, even among those who reject the significance of TC and LDL-C.
~ A Unique Twin Study On The Increased Cardiometabolic Risk In Obesity -- "Study finds that obesity, already in its early stages and independent of genetic influences, is associated with deleterious alterations in the lipid metabolism known to facilitate atherogenesis, inflammation and insulin resistance."
“The effectiveness and rapidity of statin-induced decreases in coronary events led to the speculation that statins possess cholesterol-independent effects… we describe the numerous beneficial pleiotropic effects of statins that could modulate atherogenesis.”
Trans isomers increase lipoproteina, a non-dietary-related risk of atherogenesis, to levels higher than the corresponding chain-length saturated fatty acid….
The potential involvement of extracellular histones in atherogenesis may be envisaged through several routes.
These are involved in regulating vasculogenesis and angiogenesis after ischemic injury, interactions of cells with adhesive proteins and blood vessels, proliferation of smooth muscle cells during atherogenesis, metabolism of lipoproteins and non-thrombogenic characteristics of endothelial cells
Williams KJ, Tabas I (1995) The response-to-retention hypothesis of early atherogenesis.
Uric acid not only increases the level of inflammation in the body which can promote atherogenesis and thrombogenesis, but also may increase platelet stickiness.
Although to our knowledge no previous studies have investigated the role of HSPA8 in endothelium cells apoptosis, it might protect endothelium cells against apoptosis, which is believed to be the initiating event of atherogenesis and plays a crucial role in the transition from a stable endothelialized plaque to plaque erosion and thrombosis
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